英国CU论文代写:

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然而，这不是消耗药物的唯一后果。研究人员发现，MDMA对认知过程的影响–如思维和记忆。研究已证实，摇头丸用户遭受贫穷的记忆和思考能力的过程，有人还没有尝试过的药物相比（摩根，2000）。它也被发现干扰的过程，如睡眠周期（艾伦等人，1993）。然而，将这些影响与狂喜的问题是，服用摇头丸，它不能被排除，这些人经常熬夜跳舞，也可以采取其他药物。探讨狂喜的隔离效果，考尔特等人（2002），给松鼠猴子和狒狒的剂量，他们声称是类似于那些由一些俱乐部在一个晚上的药。这种给动物服用的剂量被发现完全破坏产生神经递质多巴胺的神经元，导致类似帕金森病的情况。此外，十只动物中有2只死亡。考尔特等人（2002）认为，“一个年轻人支持这些多巴胺细胞损伤和消耗他们的储备可能在帕金森病的风险更大。”这是被广泛接受的文学评论，脑中的多巴胺会随着年龄的增长而下降；因此，服用摇头丸艾滋病老龄化进程加快和发展疾病的易感性这样的帕金森的。

英国CU论文代写:消耗药物

However, this is not the only consequence of consuming the drug. Researchers have found that MDMA has affects on the cognitive processes – such as thinking and memory. Studies have confirmed that MDMA users suffer poorer memory and impaired thought processes, compared with people who haven't tried the drug (Morgan, 2000). It has also been found to interfere with processes such as sleep cycles (Allen et al, 1993). However, the problem with ascribing these effects to ecstasy is that people who take MDMA, it can not be ruled out that these people frequently stay up all night dancing and may also take other drugs. To investigate the isolated effects of ecstasy, Ricaurte et al (2002), gave squirrel monkeys and baboons doses of the drug that they claimed were similar to those taken by some clubbers in a single night. This dose administrated to the animals was found to completely destroy neurons that produce the neurotransmitter dopamine, and led to a condition similar to Parkinson's disease. Furthermore, 2 out of the ten animals died. Ricaurte et al (2002) conclude that "A young individual who sustains injury to these dopamine cells and depletes their reserve may be at greater risk of parkinsonism." It is widely-accepted from literature reviews that brain dopamine declines with age; therefore, taking the drug Ecstasy aids to speed up the aging process and susceptibility for developing diseases such a Parkinson’s.

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